By Jane M. Grant-Kels

This amazing dermatopathology atlas emphasizes the correlation of pathological findings with medical shows and provides a reader-friendly method of the analysis and interpretation of pores and skin biopsy effects. With an abundance of colour medical and histologic pictures, and outlines of diverse dermatological illnesses and prerequisites, this resource is a must have for somebody getting ready for dermatology or pathology board assessments, or for these wanting a powerful knowing of the scientific or pathological displays of disorder.

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J Cutan Pathol 2000; 27:569. 2. English J. Lichen aureus. J Am Acad Dermatol 1985; 12:377– 378. B Superficial and deep perivascular and periadnexal (often peri-eccrine) inflammatory cell infiltrate (Fig. 13A) Band-like lymphohistiocytic infiltrate which obscures the dermoepidermal junction (Fig. 13B) 26 B B B Taylor and Heilman Hyperplastic epidermis with spongiosis (Fig. 13B) Lymphocytes extend into the epidermis (Fig. 13B) There may be mild hyperkeratosis or focal parakeratosis Differential Diagnosis: B B Differential Diagnosis: Lichen Striatus Linear Lichen Planus Secondary Syphilis Psoriasiform/ Lichenoid Drug Eruption Epidermal spongiosis may be present Spongiosis is not a feature Eosinophils do not usually feature in infiltrate Eosinophils usually prominent in inflammatory infiltrate Inflammatory infiltrate is deep and often periadnexal Inflammatory infiltrate is limited to superficial dermis Focal parakeratosis may be present Parakeratosis is not a feature Pathophysiology: B Pathophysiology: B B Superficial and deep perivascular inflammatory cell infiltrate Plasma cells present in 80% of cases (Fig.

B B B B Pruritic, erythematous, edematous papules, and plaques Vesicles and bullae common (Fig. 6) Linear lesions or pattern of contact Toxicodendron species (poison ivy, oak, sumac) common culprits Histology: SPONGIOTIC DERMATOSES B This group of disorders is characterized by the histologic finding of spongiosis. Clinical pathologic correlation is essential as several disease entities may appear indistinguishable histologically and yet be clinically distinct. Many of these disorders present with vesicles or bullae in their early or acute forms.

2. Finan MC, Schroeter AL. Cutaneous immunofluorescence study of erythema multiforme: correlation with light microscopic patterns and etiologic agents. J Am Acad Dermatol 1984; 10(3):497 –506. 3. Paul C, Wolkenstein P, Adle H, et al. Apoptosis as a mechanism of keratinocyte death in toxic epidermal necrolysis. Br J Dermatol 1996; 134(4):710–714. -HOST REACTION Clinical Presentation: B Systemic syndrome with fever and cutaneous, gastrointestinal, and hepatic manifestations 1. Lerner KG, Kao GF, Storb R, et al.

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