By Ramon L. Sanchez

Univ. of Texas, Galveston. guide and primer in dermatopathology for citizens of pathology and dermatology, and clinical scholars. so much entries comprise scientific, histology, and different details. Illustrated in halftone. Wire-spiral binding. DNLM: epidermis Diseases--pathology.

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Linear (Verrucous) Epidermal Nevus Clinical Linear epidermal nevi are usually present at birth but may develop in childhood. They present as verrucous yellow-brown papules in a linear arrangement and can be located anywhere on the body. The localized form consists of only a single linear lesion. The systemic form, however, consists of multiple lesions, often in a parallel distribution and frequently following the lines of Blashko. Linear epidermal nevi may be small, measuring only a few centimeters or extensive, covering large portions of the body (Ichthyosis hystrix).

Individual lesions heal within a few weeks, generally with little or no scarring. 4. 4. PLEVA: There is confluent parakeratosis, prominent exocytosis of lymphocytes, interface dermatitis, necrosis of keratinocytes, and extravasated RBCs. recurring over a period of several months or sometimes even years. Occasionally larger lesions, which develop into necrotic ulcers and result in scarring, develop. Rarely a form of the disease that is characterized by ulcerative lesions which may become confluent and are associated with fever and constitutional symptoms occurs.

There is also hyperkeratosis (Fig. 6). Bullous Congenital Ichthyosiform Erythroderma This condition results from an autosomal dominant defect in keratins 1 and 10. 3 28 Dermatopathology Clinical Presents at birth with erythema and mild hyperkeratosis and frequently with denuded skin. Bullae form after mechanical trauma. Early in life, blistering is replaced by generalized hyperkeratosis with accentuation of flexural involvement. Histology 3 Histologically, epidermolytic hyperkeratosis is present, and mitoses are numerous in the epidermis.

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