By Antonella Tosti, Bianca Maria Piraccini

This new commonly illustrated atlas offers a differential advisor to analysis of issues of the hair after which bargains an evidence-based critique of the choices for treatment. This consultant covers evaluate tools, in addition to the total diversity of issues of hair, from illness and problems brought on via therapy to difficulties referring to specified teams of sufferers.

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24 Pili bifurcati. 5). Photosensitivity, with mutations of the XPD, XPB or TTDA gene, is present in about 50% of cases but there is no predisposition to skin cancers. 24). Each branch has its own cuticle. Pili bifurcati may occur in association with other hair abnormalities such as pili canaliculi and monilethrix. Clinical features The condition is rare and may produce a diffuse alopecia, more evident in some areas. 26 Diffuse partial woolly hair. 27 Pili annulati: alternating light and dark bands.

1). 3 Trichorrexis invaginata. 8 Monilethrix: the hair shaft has a beaded appearence. Medulla is absent in the internodes. 5 Netherton disease: trichorrexis invaginata, ichthyosis and atopic dermatitis. 7b 25 Hair shaft disorders Ichthyosis, which may be present at birth as congenital erythrodermia, is in most cases ichthyosis linearis circumflexa, characterized by migratory serpiginous circinate plaques of scaly erythema with a double-edged scale at the advancing borders. Genetics Netherton’s syndrome has been mapped to chromosome 5q32 and the gene has been identified as SPINK5 which encodes the serine-protease inhibitor LETKI.

Seiter S, Ugurel S, Tilgen W, Reinhold U. High-dose pulse corticosteroid therapy in the treatment of severe alopecia areata. Dermatology 2001; 202:230–4. Tosti A, Piraccini BM, Pazzaglia M, Vincenzi C. 05% under occlusion in the treatment of alopecia totalis/universalis. J Am Acad Dermatol 2003; 49:96–8. 6 Androgenetic alopecia Androgenetic alopecia (AGA) is the most common form of hair loss, affecting up to 80% of men and 50% of women in the course of their life. The disease typically manifests after puberty and is evident by the age of 30.

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