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Stanson, T. L. Whiteside and Q. P. Dou, unpublished data). 3 Molecular analysis of the chain in human carcinomas In view of the evidence reviewed above that exogenous IL-2 at nM or greater concentrations inhibited growth of carcinoma cells by inducing CCA, we considered a possibility that the expressed on carcinomas and responsible for negative signaling was different from that expressed on lymphoid cells. To investigate this possibility, mRNA for the chain was isolated, reversetranscribed, amplified, and compared in carcinomas and lymphoid cells (37).

N Engl J Med 1987;316(7):379-85 96. Beutler BA, Cerami A. Recombinant interleukin 1 suppresses lipoprotein lipase activity in 3T3L1 cells. J Immunol 1985;135(6):3969-71 97. Metcalf D. Disease states induced by hemopoietic growth factor excess: their implications in medicine. Int J Cell Cloning 1990;8 Suppl 1:374-87; discussion 387-90 98. Gadient RA, Patterson PH. Leukemia inhibitory factor, Interleukin 6, and other cytokines using the GP130 transducing receptor: roles in inflammation and injury.

Stem Cells 1999;17(3):127-37 99. Brown MA, Metcalf D, Gough NM. Leukaemia inhibitory factor and interleukin 6 are expressed at very low levels in the normal adult mouse and are induced by inflammation. Cytokine 1994:6:300-09 100. Block MI, Berg M, McNamara MJ. Passive immunization of mice against D factor blocks lethality and cytokine release during endotoxemia. J Exp Med 1993;178:1085-90 101. Alexander HR, Wong GG, Doherty GM. Differentiation factor/leukemia inhibitory factor protection against lethal endotoxemia inmice: synergistic effect with interleukin 1 and tumor necrosis factor.

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