By Icon Health Publications
In March 2001, the nationwide Institutes of health and wellbeing issued the subsequent caution: "The variety of websites delivering health-related assets grows on a daily basis. Many websites supply priceless details, whereas others can have info that's unreliable or misleading." moreover, end result of the quick elevate in Internet-based info, many hours will be wasted looking out, picking out, and printing.This e-book used to be created for doctors, scholars, and contributors of most people who are looking to behavior clinical examine utilizing the main complicated instruments to be had and spending the smallest amount of time doing so.
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Additional info for Prostate Cancer - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Specifically, this study will define SNPs and haplotypes in steroid hormone metabolizing genes, genes in the IGF pathway, and related receptor proteins. These candidate genes will be resequenced in 96 cases of advanced breast cancer, and 96 cases of advanced prostate cancer, chosen from the European, African, Latino, Japanese, and Hawaiian-origin ethnic groups. Haplotype tag SNPs will be selected after genotyping of a larger number of SNPs from a collection of 768 samples from the multigenerational CEPH (Centre d'Etude du Polymorphisme Humain) pedigrees and human diversity collection, by the Whitehead Institute for Genome Research and CEPH, and will rapidly be made publicly available.
Traditional Chinese Herbal Medicines (TCHM) have been shown to block recurrent prostate cancer growth. In our preliminary study, using a newlydeveloped androgen receptor (AR) and androgen receptor co-activator (ARA)-mediated transactivation assay, it was found that some extracts from TCHM inhibited AR-induced gene activation, suggesting TCHM is an excellent source to search for new antiandrogen drugs. A cell-based androgen/AR activation assay developed from this study is advantageous in discovering antiandrogens that exert their inhibitory function at different stages of an androgen-induced gene activation.
The exact biological basis for this, however, is unknown. One possible explanation is that calcium can reduce the body's level of 1,25-vitamin D3 which has been shown to protect the prostate, but various scientists have questioned the significance of this concept. Understanding the molecular properties of calcium channels, including their regulation by 1,25-vitamin D3, may help explain the role of the nutrient calcium in prostate cancer development. Calcium channels play a important roles in the metabolism of prostate cancer epithelial cells but little is known about their implications in prostate cancer development.
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