By Sanjay Ghosh

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18 Recent Advances in Dermatology Contd... TLR5 • Flagellin binds with TLR5 and leads to elaborate cytokines like TNF-α Melanoma TLR7 • TLR7 helps mounting protecting immune response against melanoma and arresting disease progression Non-melanoma Skin Cancers TLRs 1, 2, 3, 5, 6, 7, 8 • SCC but not BCC showed significantly elevated mRNA expression of TLRs 1,2,3,5,6,7,8, TRIF and TRAF1 • BCC shows increased IFN-g TLR7, TLR8 • TLR 7 and TLR8 have been employed to activate the proinflammatory machinery against BCC and SCC SLE Sjogren’s Syndrome TLR7 • TLR7 promotes elaboration of INF-α TLR8 • Its ligand can induce production of Th1 cytokines.

82. Seung NR, Park EJ, Kim CW, et al. Comparison of expression of heat-shock protein 60, Toll-like receptors 2 and 4, and T-cell receptor γδ in plaque and guttate psoriasis. J Cutan Pathol. 2007;34:903–11. 83. Ohashi K, Burkart V, Flohe S, et al. Cutting edge: heat shock protein 60 is a putative endogenous ligand of the Tolll-like receptors-4 complex. J Immunol. 2000;164:558. 84. Zanin-Zhorov A, Nussbaum G, Franitza S, et al. T cells respond to heat shock protein 60 via TLR2: activation of adhesion and inhibition of chemokine receptors.

2008;52:477–82. 1 A third of these patients require specialist treatment due to severity of disease or nonresponse to treatment. It is ‘chronic’ in nature,2 and has the ability to cause cosmetic and psychologic disaster. Follicular cornification, sebaceous hyperplasia, Propionibacterium acnes proliferation and associated inflammation have been known to be the pathogenic pillars of acne. Traditionally believed sequence of events in acne pathogenesis is formation of a keratin plug (microcomedone), androgen mediated activation of sebaceous glands, P.

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